National Academies Press: OpenBook

Asbestos: Selected Cancers (2006)

Chapter: APPENDIX E Case-Control Results Tables

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Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

APPENDIX E
Case-Control Results Tables

TABLE E.1 Pharyngeal Cancer and Exposure to Asbestos—Case-Control Studies

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Berrino et al. 2003

100 male cases of hypopharyngeal cancer from six centers in Southern Europe, < 55 years old (adjusted for smoking and alcohol consumption)

 

 

 

Possible

na

1.8 (0.9-3.9)

 

Probable

na

1.8 (0.6-5.0)

 

(More detailed findings from combined analysis with 215 cases of laryngeal cancer on Table E.2)

 

 

Luce et al. 2000

5 hypopharyngeal cancer cases among residents of New Caledonia

 

 

 

Whitewash from tremolite asbestos

1

0.64 (0.01-6.68)

Marchand et al. 2000

206 hypopharyngeal cancer cases among male residents of six cities in France (adjusted for smoking and alcohol consumption)

 

 

 

Any exposure

161

1.80 (1.08-2.99)

 

Low cumulative exposure

52

1.92 (1.03-3.57)

 

Intermediate

52

1.40 (0.74-2.63)

 

High

57

2.14 (1.14-4.01)

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Gustavsson et al. 1998

138 pharyngeal cancer cases among male residents of two regions in Sweden (adjusted for smoking and alcohol consumption)

 

 

 

Asbestos (low)

24

1.01 (0.57-1.80)

 

Asbestos (high)

22

1.08 (0.62-1.91)

Zheng et al. 1992b

115 male oral or pharyngeal cancer cases among residents of Shanghai, China Asbestos, occupational exposure

16

1.81 (0.91-3.60)a

Merletti et al. 1991

86 oral cavity or oropharynx (n = 12) cancer cases among male residents of Turin, Italy (adjusted for smoking and alcohol consumption)

 

 

 

Any exposure

45

1.1 (na)

 

Probable or definite

3

0.4 (na)

NOTES: CI = Confidence interval; na = not available; RR = relative risk. Data points included in meta-analyses are bolded.

* Full citations can be found in the reference list for Chapter 6.

aOR and 95% CI calculated with standard methods from observed numbers of exposed cases and controls in original paper.

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

TABLE E.2 Laryngeal Cancer and Exposure to Asbestos—Case-Control Studies

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Berrino et al. 2003

213 male cases of endolaryngeal cancer from six centers in Southern Europe, < 55 years old

 

 

 

Possible

na

1.7 (1.0-3.0)

 

Probable

na

1.8 (0.8-4.0)

 

Combined analysis with 100 hypopharyngeal cancer cases

 

 

 

Asbestos (JEM-derived agent), any exposure

215

1.6 (1.0-2.5)

 

10+ years duration and 20+ years lag

121

1.4 (0.8-2.4)

 

Likelihood of exposure

 

 

 

Possible

175

1.7 (1.1-2.8)

 

Probable

40

1.9 (0.9-3.8)

 

Duration of exposure

 

 

 

< 10 years

na

1.3 (0.6-2.7)

 

10-19 years

na

1.4 (0.7-2.7)

 

≥ 20 years

na

1.7 (0.9-3.0)

p-trend > 0.05

 

Tertiles of weighted exposure

 

 

 

1

na

1.4 (0.8-2.3)

 

2

na

1.9 (1.2-3.2)

 

3

na

1.6 (1.0-2.6)

p-trend = 0.037

Dietz et al. 2003

257 laryngeal cancer cases among residents of Rhein-Neckar region, Germany Asbestos

59

1.3 (0.8-2.1)

Elci et al. 2002

940 laryngeal cancer cases among male residents of Istanbul, Turkey (smoking-adjusted)

 

 

 

Asbestos (JEM-derived agent)

150

1.0 (0.8-1.3)

 

Glottis

28

0.8 (0.5-1.2)

 

Supraglottis

71

1.0 (0.8-1.4)

 

Other laryngeal

51

1.2 (0.9-1.7)

 

Intensity of exposure

 

 

 

Low

45

0.9 (0.6-1.3)

 

Medium

93

1.2 (0.9-1.6)

 

High

12

0.6 (0.3-1.1)

 

Probability of exposure

 

 

 

Low

121

1.2 (0.9-1.5)

 

Medium

20

0.6 (0.4-1.1)

 

High

9

0.7 (0.3-1.5)

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Luce et al. 2000

20 laryngeal cancer cases among male residents of New Caledonia (all smokers)

 

 

 

Whitewash from tremolite asbestos

3

0.72 (0.22-2.30)

 

Melanesians

2

0.71 (0.14-3.63)

 

Non-Melanesians

1

0.60 (0.07-5.22)

Marchand et al. 2000

296 laryngeal cancer cases among male residents of six cities in France (smoking-adjusted)

 

 

 

Any exposure

216

1.24 (0.83-1.90)

 

Low cumulative exposure

67

1.10 (0.66-1.82)

 

Intermediate

72

1.20 (0.73-1.99)

 

High

77

1.47 (0.87-2.46)

 

Supraglottic, any exposure

56

1.12 (0.61-2.05)

 

Low cumulative exposure

15

0.84 (0.38-1.84)

 

Intermediate

22

1.31 (0.62-2.76)

 

High

19

1.27 (0.58-2.78)

 

Glottic and subglottic, any exposure

75

1.15 (0.68-1.95)

 

Low cumulative exposure

27

1.19 (0.62-2.27)

 

Intermediate

21

0.90 (0.45-1.78)

 

High

27

1.44 (0.73-2.83)

 

Epilarynx, any exposure

77

1.77 (0.94-3.30)

 

Low cumulative exposure

22

1.45 (0.67-3.13)

 

Intermediate

25

1.69 (0.79-3.64)

 

High

30

2.22 (1.05-4.71)

De Stefani et al. 1998

112 laryngeal cancer cases among male residents of Montevideo, Uruguay (smoking-adjusted)

 

 

 

Asbestos (self-reported agent)

 

231.8 (0.9-3.2)

 

1-20 years

4

0.9 (0.3-2.7)

 

20+ years

19

2.4 (1.2-4.8)

 

Supraglottic

na

2.3 (0.9-5.7)

 

Glottic

na

2.9 (0.8-10.5)

Gustavsson et al. 1998

157 laryngeal cancer cases among male residents of two regions in Sweden

 

 

 

Asbestos (low)

28

1.21 (0.73-2.02)

 

Asbestos (high)

34

1.69 (1.05-2.74)

 

Quartile I

13

1.16 (1.02-1.32)

 

Quartile II

15

1.35 (1.04-1.74)

 

Quartile III

16

1.56 (1.06-2.30)

 

Quartile IV

18

1.82 (1.08-3.04)

p-trend = 0.02

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Muscat and Wynder 1992

186 laryngeal cancer cases among white, male residents of New York, Illinois, Michigan, and Pennsylvania, US

 

 

 

Asbestos, any exposure

66

1.1 (0.7-1.9)

 

Glottis

40

1.3 (0.7-2.7)

 

Supraglottis

26

1.1 (0.5-2.6)

Wortley et al. 1992

235 laryngeal cancer cases among residents of western Washington state, US

 

 

 

Asbestos—peak

 

 

 

None

145

1.0

 

Low

3

1.2 (0.6-7.1)

 

Medium

57

1.3 (0.8-2.0)

 

High

30

1.1 (0.6-1.9)

 

Asbestos—duration

 

 

 

< 1 year

151

1.0

 

1-9

50

1.0 (0.5-2.1)

 

≥ 10

34

1.2 (0.6-2.3)

 

Asbestos—exposure scores

 

 

 

< 5

173

1.0

 

5-19

25

1.1 (0.6-2.1)

 

≥ 20

37

1.4 (0.7-2.5)

Zheng et al. 1992a

201 laryngeal cancer cases among residents of Shanghai, China (smoking-adjusted)

 

 

 

Asbestos, occupational exposure

26

2.0 (1.0-4.3)

Ahrens et al. 1991

85 laryngeal cancer cases among male residents of Bremen, Germany (smoking-adjusted)

 

 

 

Asbestos

na

1.1 (0.5-2.4)

Brown et al. 1988

180 laryngeal cancer cases among male residents along Gulf Coast of Texas (smoking-adjusted)

 

 

 

Asbestos

88

1.5 (1.0-2.2)

 

< 5 years

20

1.3 (0.7-2.6)

 

5-14

24

2.2 (1.1-4.3)

 

≥ 15

40

1.4 (0.8-2.4)

 

unknown

4

 

Zagraniski et al. 1986

92 laryngeal cancer cases among white, male residents of New Haven, CT (smoking-adjusted)

 

 

 

Asbestos workers (ever held occupation)

11

1.1 (0.4-2.9)

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

Reference

Study Population

Exposed Cases

Estimated RR (95% CI)

Olsen and Sabroe 1984

276 male laryngeal cancer cases among residents of Denmark (smoking-adjusted) Asbestos

17

1.8 (1.0-3.4)

Burch et al. 1981

184 laryngeal cancer cases among male residents of southern Ontario, Canada (smoking-adjusted)

 

 

 

Self-reported asbestos exposure

36

1.6 (p = 0.069)

 

Occupational hygienist classified exposure

14

2.3 (p = 0.052)

Hinds et al. 1979

47 laryngeal cancer cases among male residents of three counties in WA; self-reported asbestos exposure

 

 

 

All subtypes

25

1.75 (p = 0.21)

 

Glottis

na

1.29 (p = 0.63)

 

Supraglottis

na

4.00 (p = 0.22)

Shettigara and Morgan 1975

43 laryngeal cancer cases among male hospital patients in Toronto, Canada Asbestos

10

(0 exposed controls)

Stell and McGill 1973

100 laryngeal cancer cases among male hospital patients in Liverpool, UK Asbestos

31

14.53 (4.27-49.43)a

NOTES: CI = Confidence interval; na = not available; RR = relative risk. Data points included in meta-analyses are bolded.

* Full citations can be found in the reference list for Chapter 6.

aOR and 95% CI calculated with standard methods from observed numbers of exposed cases and controls in original paper.

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

TABLE E.3 Esophageal Cancer and Exposure to Asbestos—Case-Control Studies

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Parent et al. 2000

99 esophageal cancer cases among male residents of Montreal, Canada; IH-derived agent: chrysotile asbestos (smoking-adjusted)

 

 

 

All subtypes

 

 

 

Any exposure

21

1.4 (0.8-2.4)

 

Nonsubstantial

19

1.4 (0.8-2.5)

 

Substantial

2

1.3 (0.3-6.2)

 

63 squamous-cell carcinomas

 

 

 

Any exposure

17

2.0 (1.1-3.8)

 

Nonsubstantial

16

2.1 (1.1-4.0)

 

Substantial

1

1.1 (0.1-9.7)

Gustavsson et al. 1998

122 esophageal cancer cases among male residents of two regions in Sweden; IH-derived agent (smoking-adjusted)

 

 

 

Asbestos (low)

22

1.21 (0.67-2.17)

 

Asbestos (high)

21

1.00 (0.54-1.82)

Hillerdal 1980

Gastrointestinal carcinoma cases among male residents of Uppsala county, Sweden (exposure = pleural plaques)

 

 

 

21 esophageal

1

2.86 (0.07-15.91)a

NOTES: CI = Confidence interval; na = not available; RR = relative risk. Data points included in meta-analyses are bolded.

* Full citations can be found in the reference list for Chapter 6.

aOR and 95% CI calculated with standard methods from observed and expected numbers presented in original paper.

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

TABLE E.4 Stomach Cancer and Exposure to Asbestos—Case-Control Studies

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Krstev et al. 2005

443 stomach cases among residents of Warsaw, Poland

 

 

 

285 males, ever exposed

42

1.5 (0.9-2.4)

 

1-9 years

19

1.2 (0.6-2.3)

 

10 years

23

1.9 (0.9-3.8)

 

158 females, ever exposed

1

0.3 (0.03-3.0)

 

1-9 years

1

0.4 (0.0-6.0)

 

10 years

0

Ekstrom et al. 1999

565 gastric cancer cases among residents of Sweden

155

1.11 (0.87-1.42)

Parent et al. 1998

250 male gastric cancer cases among residents of Montreal, Canada

 

 

 

Chrysotile asbestos

 

 

 

Nonsubstantial

43

1.2 (0.8-1.7)

 

Substantial

4

0.7 (0.2-1.8)

 

Amphibole asbestos

 

 

 

Nonsubstantial

10

0.6 (0.3-1.2)

 

Substantial

3

1.9 (0.6-6.9)

Cocco et al. 1994

640 gastric cancer cases among male residents of Italy

 

 

 

Ever exposed

239

0.7 (0.5-1.1)

 

21+ years

na

1.4 (0.6-3.0)

Hillerdal 1980

Gastrointestinal carcinoma cases among male residents of Uppsala county, Sweden (exposure = pleural plaques)

 

 

 

148 stomach

6

2.40 (0.88-5.22)a

NOTES: CI = Confidence interval; na = not available; RR = relative risk. Data points included in meta-analyses are bolded.

* Full citations can be found in the reference list for Chapter 6.

a95% CI calculated with standard methods from observed and expected numbers presented in original paper.

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

TABLE E.5 Colorectal Cancer and Exposure to Asbestos—Case-Control Studies

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Goldberg et al. 2001

497 colon cancer cases among male residents of Montreal, Canada; industrial-hygiene-derived agent

 

 

 

Adjusted for age and non-occupational factors

 

 

 

Nonsubstantial

60

0.9 (0.6-1.3)

 

Substantial

18

2.1 (1.1-4.0)

 

Further adjusted for occupational factors

 

 

 

Nonsubstantial

60

0.9 (0.6-1.3)

 

Substantial

18

1.8 (0.9-3.6)

 

Frequency

 

 

 

1-5%

21

0.9 (0.5-1.6)

 

6-30%

49

1.1 (0.7-1.5)

 

> 30%

8

1.5 (0.6-3.7)

 

Concentration

 

 

 

Low

40

0.9 (0.6-1.4)

 

Medium

32

1.2 (0.8-1.8)

 

High

6

1.4 (0.4-4.3)

 

Duration (10-year increment)

78

1.1 (0.9-1.2)

Dumas et al. 2000

257 rectal cancer cases among male residents of Montreal, Canada

 

 

 

Chrysotile, any

30

0.7 (0.5-1.0)

 

Substantial

3

0.5 (0.2-1.6)

 

Amphiboles, any

11

0.7 (0.3-1.2)

 

Substantial

2

1.5 (0.3-7.6)

Demers et al. 1994

261 colorectal cancer cases among white males residents of southeast Michigan

15

0.5 (0.3-1.0)

 

Duration (years)

 

 

 

< 20

9

0.6 (0.3-1.5)

 

20+

6

0.4 (0.1-1.2)

 

Latency (years)

 

 

 

< 40

5

0.4 (0.1-1.3)

 

40+

10

0.6 (0.2-1.4)

Vineis et al. 1993

74 colon cancer cases among male residents of industrialized northern Italy (job titles) Jobs with putative asbestos exposure

4

4.8 (1.05-21.5)

Garabrant et al. 1992

419 male colon cancer cases among residents of Los Angeles County, CA Never exposed

353

1.00

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

 

No latency

 

 

 

Any exposure

66

0.99 (0.66-1.50)

 

Asbestos on hands and clothes

 

 

 

Did not get on hands or clothes

17

2.32 (0.87-6.23)

 

Got on hands or clothes

49

0.82 (0.52-1.30)

 

Use of mask

 

 

 

Did not wear mask

55

0.95 (0.61-1.46)

 

Wore mask

11

1.43 (0.49-4.17)

 

Frequency of exposure

 

 

 

< 5 times/week

18

1.00 (0.50-2.00)

 

5 times/week

31

0.79 (0.43-1.46)

 

Brief, intense exposure

17

1.48 (0.64-3.38)

 

Ordinal trend

 

p = 0.70

 

Duration of exposure (years)

 

 

 

< 5

24

0.98 (0.53-1.84)

 

5-14

20

1.47 (0.67-3.22)

 

≥ 15

22

0.76 (0.39-1.49)

 

Continuous trend

 

p = 0.61

 

Ordinal trend

 

p = 0.81

 

Time since first exposure (years)

 

 

 

< 1-14

10

1.66 (0.54-5.10)

 

15-29

21

1.37 (0.65-2.91)

 

≥ 30

35

0.77 (0.45-1.31)

 

Continuous trend

 

p = 0.61

 

Ordinal trend

 

p = 0.66

 

Cumulative exposure index

 

 

 

1-30

41

1.26 (0.74-2.15)

 

31-60

11

0.80 (0.34-1.88)

 

61

14

0.65 (0.28-1.51)

 

Continuous trend

 

p = 0.22

 

Ordinal trend

 

p = 0.46

 

15-year latency

 

 

 

Exposed, latency > 15 years

56

0.93 (0.60-1.44)

 

Asbestos on hands and clothes

 

 

 

Did not get on hands or clothes

12

1.75 (0.62-4.94)

 

Got on hands or clothes

44

0.83 (0.51-1.33)

 

Use of mask

 

 

 

Did not wear mask

46

0.86 (0.55-1.37)

 

Wore mask

10

1.95 (0.55-6.90)

 

Frequency of exposure

 

 

 

< 5 times/week

14

0.83 (0.39-1.76)

 

≥ 5 times/week

30

0.93 (0.49-1.77)

 

Brief, intense exposure

12

1.14 (0.46-2.87)

 

Ordinal trend

 

p = 0.78

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

Reference

Study Population

Exposed Cases

Estimated RR (95% CI)

 

Duration of exposure (years)

 

 

 

< 5

19

0.74 (0.37-1.47)

 

5-14

21

1.60 (0.75-3.44)

 

≥ 15

16

0.69 (0.30-1.55)

 

Continuous trend

 

p = 0.58

 

Ordinal trend

 

p = 0.79

 

Cumulative exposure index

 

 

 

1-30

36

1.07 (0.63-1.81)

 

31-60

10

0.94 (0.33-2.65)

 

≥ 61

10

0.55 (0.21-1.47)

 

Continuous trend

 

p = 0.33

 

Ordinal trend

 

p = 0.40

Gerhardson de Verdier et al. 1992

Colon and rectal cancer cases among male residents of Stockholm, Sweden; self-reported agents

 

 

 

163 colon cancers

22

1.9 (0.9-4.2)

 

Right colon

16

2.6 (1.2-5.9)

 

Left colon

3

0.5 (0.1-1.9)

 

107 rectal cancers

17

1.9 (0.8-4.6)

 

Colorectal cancer: latency (years)

 

 

 

1-19

5

1.4 (0.3-9.9)

 

20+

34

2.0 (1.0-3.9)

 

1-29

12

1.6 (0.5-5.0)

 

30+

27

2.0 (1.0-4.4)

 

1-39

22

1.4 (0.7-3.0)

 

40+

17

3.2 (1.1-11.5)

Neuget et al. 1991

51 colorectal cancer cases among males undergoing colonoscopy in 3 NYC medical centers

 

 

 

Asbestos exposure

10

1.8 (0.8-5.6)

 

Significant exposure

3

4.3 (0.8-23.5)

Fredriksson et al. 1989

329 colon cancer cases among residents of Umea, Sweden

 

 

 

Asbestos, low grade

na

1.2 (0.6-2.4)

 

Asbestos, high grade

na

2.1 (0.8-5.8)

Spiegelman and Wegman 1985

Colorectal cancer cases in seven US metropolitan areas and two states; JEM-derived agent

 

 

 

Males: 343 colorectal cancer

na

1.28 (p = 0.17)

 

224 colon cancer only

na

1.22 (p = 0.33)

 

Females: 208 colorectal cancer

na

1.08 (p = 0.65)

 

171 colon cancer only

na

1.09 (p = 0.64)

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×

Reference*

Study Population

Exposed Cases

Estimated RR (95% CI)

Hardell 1981

153 colon cancer cases among male residents of Umea, Sweden Asbestos, any

16

1.9 (1.0-3.6)

Hillerdal 1980

Gastrointestinal carcinoma cases among male residents of Uppsala county, Sweden (exposure = pleural plaques)

 

 

 

108 colon

3

1.67 (0.34-4.87)a

 

101 rectal

3

1.76 (0.36-5.16)a

NOTES: CI = Confidence interval; na = not available; RR = relative risk. Data points included in meta-analyses are bolded.

* Full citations can be found in the reference list for Chapter 6.

aOR and 95% CI calculated with standard methods from observed and expected numbers presented in original paper.

Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×
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Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×
Page 298
Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×
Page 299
Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
×
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Suggested Citation:"APPENDIX E Case-Control Results Tables." Institute of Medicine. 2006. Asbestos: Selected Cancers. Washington, DC: The National Academies Press. doi: 10.17226/11665.
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Next: APPENDIX F Initial Analyses of Available Data Concerning Cancers of Colon and/or Rectum and Asbestos Exposure »
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In conjunction with drafting comprehensive legislation concerning compensation for health effects related to asbestos exposure (the Fairness in Asbestos Injury Act), the Senate Committee on the Judiciary directed the Institute of Medicine to assemble the Committee on Asbestos: Selected Health Effects. This committee was charged with addressing whether asbestos exposure is causally related to adverse health consequences in addition to asbestosis, mesothelioma, and lung cancer. Asbestos: Selected Cancers presents the committee's comprehensive distillation of the peer-reviewed scientific and medical literature regarding association between asbestos and colorectal, laryngeal, esophageal, pharyngeal, and stomach cancers.

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