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Summary
Pages 1-6

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From page 1...
... In planning this workshop, the Roundtable on Environmental Health Sciences, Research, and Medicine wanted to address the link between environmental factors and the development of cancer in the light of recent advances in genomics and, more specifically, in toxicogenomics and gene-environment interactions. Speakers were invited from many scientific discliplines including epidemiology, molecular biology, oncology, microbiology and immunology, nutrition science, and human genetics.
From page 2...
... He discussed the findings that both essential and nonessential dietary nutrients can markedly influence several key biological events, including cell cycle regulation, processes involved in replication or transcription, immunocompetence, and factors involved in apoptosis, or programmed cell death. These observations suggest that specific foods or components may have the potential to markedly reduce cancer risk.
From page 3...
... Anderson Cancer Center, described how progress toward preventing, diagnosing, and treating cancer will be hampered by the nation's inability to deal effectively with the greater cancer burden borne by certain vulnerable populations. These populations are typically defined as groups at higher-than-average risk of death, disease, and disability, and include people with low incomes, low literacy rates, the elderly, those in rural communities, African Americans, Hispanics, American Indians and Alaska Natives, and other ethnic minorities.
From page 4...
... Understanding the role of environment in breast cancer is an area of ongoing research. Brian Henderson, University of Southern California; Mary Wolff, Mount Sinai School of Medicine; and Olufunmilayo Olopade, University of Chicago, discussed studies under way to address how complex genetic factors or hormonal milieu may alter environmental risk factors.
From page 5...
... Moreover, the international and racial-ethnic variations in prostate cancer incidence, combined with the effects of migration on risk patterns, suggest that gene-environment interaction may be involved in determining prostate cancer risk. Donald Coffey, Johns Hopkins University, and Robert DiPaola, Cancer Institute of New Jersey, discussed the relative roles of diet, nutritional supplements, and hormones in risks for prostate cancer.


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