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SECONDARY ETIOLOGICAL FACTORS IN THE CAUSATION OF MOTION SICKNESS
Pages 83-88

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From page 83...
... Although individual differences in sensitivity of the nonauditory labyrinth undoubtedly constitute the primary factor involved, extralabyrinthine influences constitute secondary etiologic factors of importance under certain circumstances. Vision, cerebral activity, olfaction, food, ambient air temperature, sexual differences, age, and chemical toxicity including alcohol, illness, and adaptation are discussed in relation to their influence on motion-sickness susceptibility.
From page 84...
... Rev., in press) who stated -- It seems likely that vision modifies vestibular responses normally, in the sense that olfaction modifies salivation, and it may be that a visual stimulus which is normally associated with a vestibular stimulus would give rise to a central vestibular activity in the absence of the vestibular stimulus.
From page 85...
... Similar experiences have been reported by pilots as the result of so-called Bunting aircraft maneuvers with resulting projectile expulsion of the gastric contents, but without any accompanying nausea. This response cannot be considered as true motion sickness, but it may well account for the difference of opinion that exists as to food in the stomach and susceptibility to motion sickness.
From page 86...
... An additional effect that undoubtedly summates with such increased vestibular sensitivity comes from the effect of alcohol's inducing nausea and vomiting even in the absence of motion. These effects of alcohol can thus add up to a vicious long-lasting combination causing greatly increased sensitivity, the degree of which is dependent upon both the amount of alcohol involved and the subject's sensitivity to alcohol and motion.
From page 87...
... H.; AND YOUNG, F A.: The Differential Effects of a Rotary Visual Field on Susceptibles and Nonsusceptibles to Motion Sickness.


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