Biographical Memoirs Volume 71 (1997) / Chapter Skim
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JEROME W. CONN
JEROME W. CONN
Pages 1-16
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13io,qraphicat Memoirs VOLUME 7 1
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the new knowlecige in hormone structure that arose in the micicIle clecacles of this century to the definition of important clinical syndromes of hormone excess en c! cleficiency.
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from the University of Michigan School of Medicine in 1932 with honors en c! as a member of the Alpha Omega Alpha Honor Society.
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This was quite an achievement in view of the clifficulty of obese patients to reach normal weight by clietary restriction. Aldosterone and the Regulation of Salt Excretion.
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Metabolism in 1943. Because acclimatization to tropical heat was a major military concern in the South Pacific, Jerry undertook a series of investigations of the regulation of salt loss in sweat in conscientious objectors exposer!
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Jerry took the occasion of his presiclential aciciress to the Central Society for Clinical Research on October 29, 1954, to present for the first time his extensive clinical investigations of this new syndrome, which he caller! primary aTclosteronism.
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cortisone administration. This stress to insulin secretory capacity proved quite effective in predicting which members of a diabetic family were most likely to come clown with the clinical disease.
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They wish eventually to reach the standing and respect in clinical research which they believe you have achieved, and many of them will, and perhaps to a greater degree! Let us set for them a proper example of kindness, friendliness, and common decency.
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, the Banting Mecial of the American Diabetes Association (1958) , the Henry Russel Award of the University of Michigan, the Gorclon Wilson Mecial of the American Clinical en c!
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tennis games with young members of his laboratory with the same intense competitive spirit as he clic! his meclical research.
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199 :555-64. 1949 Electrolyte composition of sweat: Clinical implications as an index of adrenal cortical function.
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Conn. Primary aldosteronism versus hypertensive disease with secondary aldosteronism.
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Primary reninism. Hypertension, hyperreninemia, and secondary aldosteronism due to repin-producing juxtaglomerular cell tumors.
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